Age, Biography and Wiki

Collin Y. Ewald was born on 1980 in Basel, is a Swiss molecular biologist. Discover Collin Y. Ewald's Biography, Age, Height, Physical Stats, Dating/Affairs, Family and career updates. Learn How rich is he in this year and how he spends money? Also learn how he earned most of networth at the age of 44 years old?

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Age 44 years old
Zodiac Sign
Born 1980, 1980
Birthday 1980
Birthplace Basel
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We recommend you to check the complete list of Famous People born on 1980. He is a member of famous with the age 44 years old group.

Collin Y. Ewald Height, Weight & Measurements

At 44 years old, Collin Y. Ewald height not available right now. We will update Collin Y. Ewald's Height, weight, Body Measurements, Eye Color, Hair Color, Shoe & Dress size soon as possible.

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Dating & Relationship status

He is currently single. He is not dating anyone. We don't have much information about He's past relationship and any previous engaged. According to our Database, He has no children.

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Collin Y. Ewald Net Worth

His net worth has been growing significantly in 2023-2024. So, how much is Collin Y. Ewald worth at the age of 44 years old? Collin Y. Ewald’s income source is mostly from being a successful . He is from . We have estimated Collin Y. Ewald's net worth, money, salary, income, and assets.

Net Worth in 2024 $1 Million - $5 Million
Salary in 2024 Under Review
Net Worth in 2023 Pending
Salary in 2023 Under Review
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1980

Collin Yvès Ewald (born 1980 in Basel) is a Swiss scientist investigating the molecular mechanisms of healthy aging.

He is a molecular biologist and a professor at ETH Zurich, where he leads the Laboratory of Extracellular Matrix Regeneration.

His research focuses on the remodeling of the extracellular matrix during aging and upon longevity interventions.

Collin Ewald was educated at Mathematisch-Naturwissenschaftliches Gymnasium (MNG) Basel, Switzerland.

After completing his bachelor in molecular biology at the University of Basel, he joined the labs of Joy Alcedo and Nancy Hynes studying the function of a breast cancer metastasis gene Memo1 in the model organism C. elegans at the Friedrich Miescher Institute (FMI) for Biomedical Research, Basel, Switzerland.

In the Alcedo lab, he became interested in how neurons regulate aging and went on to do a Ph.D. in Neuroscience at the Graduated Center of the City University of New York, USA.

Mentored by his Ph.D. supervisor Chris Li, he discovered a conserved genetic link between Insulin/IGF-1 signaling and Alzheimer's disease amyloid precursor protein (APP) orthologues.

To deepen his knowledge in aging research, he joined T. Keith Blackwell's lab as a postdoctoral fellow at Harvard Medical School, unraveling the importance of Insulin/IGF-1 signaling in promoting collagen homeostasis during longevity.

2013

He is named under the top 15 Longevity Influencer in Switzerland and world influencer ETH domain, Who is Who in Medical Research, is in the top 0.5% of the worldwide longevity experts, and has received multiple awards, including the Ellison Medical Foundation and American Federation for Aging Research fellowship in 2013, the DeLill Nasser Award in 2015, and the SNSF Professorship in 2016.

2015

In 2015, he became a Junior Faculty Member at the Joslin Diabetes Center, Instructor in Medicine at Harvard Medical School, and visiting scholar at the Whitehead Institute (Massachusetts Institute of Technology).

2016

After ten years in the US, in 2016, he secured the SNSF professorship to return to Switzerland to join the Institute for Translational Medicine.

as an assistant professor at ETH Zürich.

To be at the forefront and to interconnect the two research fields of aging and matrix biology, he founded the Swiss Society for Aging Research, is the vice president of the German Society of Aging Research, and he re-established the Swiss Society for Matrix Biology.

He is an independent scientific advisor of the longevity-start-up-company-builder Maximon AG, and a co-founder of Avea Life AG.

His research centers around the remodeling of the extracellular matrix, ensuring tissue and cellular homeostasis during healthy aging.

In collaboration with Alexandra Naba, he defined the proteins outside of cells forming the extracellular matrix, the so-called matrisome of C. elegans.

Over ten thousand phenotypes stem from mutations in matrisome genes in humans and across species.

He coined the term matreotype that is the extracellular matrix composition caused or associated by a cellular or physiological status, genotype, or phenotype.

Using gene expression data from humans at different ages and tissues, his team defined the youthful matreotype and used it to predict drugs that slow aging.

His research group also showed that even close to the end of an individual's life, it is possible to double the lifespan of C. elegans.