Age, Biography and Wiki
Harald Jockusch was born on 1939, is a German biologist and artist. Discover Harald Jockusch's Biography, Age, Height, Physical Stats, Dating/Affairs, Family and career updates. Learn How rich is he in this year and how he spends money? Also learn how he earned most of networth at the age of 85 years old?
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85 years old |
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1939 |
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1939 |
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We recommend you to check the complete list of Famous People born on 1939.
He is a member of famous model with the age 85 years old group.
Harald Jockusch Height, Weight & Measurements
At 85 years old, Harald Jockusch height not available right now. We will update Harald Jockusch's Height, weight, Body Measurements, Eye Color, Hair Color, Shoe & Dress size soon as possible.
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Dating & Relationship status
He is currently single. He is not dating anyone. We don't have much information about He's past relationship and any previous engaged. According to our Database, He has no children.
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Harald Jockusch Net Worth
His net worth has been growing significantly in 2023-2024. So, how much is Harald Jockusch worth at the age of 85 years old? Harald Jockusch’s income source is mostly from being a successful model. He is from . We have estimated Harald Jockusch's net worth, money, salary, income, and assets.
Net Worth in 2024 |
$1 Million - $5 Million |
Salary in 2024 |
Under Review |
Net Worth in 2023 |
Pending |
Salary in 2023 |
Under Review |
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Source of Income |
model |
Harald Jockusch Social Network
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Timeline
Harald Jockusch (born 1939 in Frankfurt am Main) is a German biologist and artist with the alias Hal Jos.
During the analysis of the host defence discovery of temperature sensitive (ts) ts TMV coat proteins of tobacco mosaic Virus (TMV)
Relationship between the structural stability and amino acid replacements of mutant TMV coat proteins (collaboration with H.-G. Wittmann and Brigitte Wittmann-Liebold)
More recent work: ts TMV coat proteins as models for misfolded (toxic) proteins in the host plant cell and in transfected animal cells
Synthesis of active phage Q beta replicase in a cell-free system
A mouse mutant, the so-called ADR mouse, was characterized as genetic model for human myotonia type Becker.
As in the human Becker and Thomsen myotonias, a strongly reduced chloride conductance of the muscle fibre membrane causes hyperexcitability of the muscle fibre, leading to episodic stiffness.
In the ADR mouse the mutated gene was identified as coding for the then newly discovered muscular chloride channel (in collaboration with the group of Thomas Jentsch).
These results lead to the identification of the human myotonia gene mutations in which either cause recessive Becker or dominant Thomsen myotonia depending on the change in the amino acid sequence.
Biomechanical analyses of the consequence of the deficiencies in the cytoskeletal proteins dystrophin (the protein deficient in Duchenne muscular dystrophy) and desmin in isolated muscle fibres and muscle cell cultures
Biochemical investigations into the neurodegeneration using the wobbler mouse model for ALS.
The responsible gene was identified as Vps54, a gene involved in protein sorting during the retrograde vesicle transport within the cell (in collaboration with the group of Miriam Meisler, Ann Arbor)
Discovery of a feedback mechanism between the extracellular protease ADAM 8 and the inflammation signalling molecule tumour necrosis factor alpha (TNFalpha) (with Jörg-Walter Bartsch)
Proteomics of tissues affected by hereditary neuromuscular diseases in the mouse model; analysis of the testis in the wobbler ALS mouse model
Morphogenesis of cardiac and skeletal muscle and of the pancreas
Topology of the metazoan body plan
Dynamics of frequency of family names after imposing the rule "rare wins"